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The Ca2+ -independent transient outward K+ current (Ito) plays a crucial role in shaping the cardiac action potential. In the present study, we examined whether angiotensin II (AngII) regulated the Ito as well as the putative intracellular cascade responsible for the effects.

Interaction of angiotensin II with the angiotensin type 2 receptor inhibits the cardiac transient outward potassium current