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arterial hypertension and her family physician prescribed irbesartan 150 mg daily. In the following months, the patient perceived a significant reduction of headache attacks. However, in January 2003 the patient developed an attack of migraine aura without headache. She reported symptoms of a scintillating scotoma, gradually expanding across the right visual field and lasting for approximately 30 min. The aura was not followed by headache. The patient had seven additional attacks in a two-year period, with the same clinical features. All the attacks fulfilled the ICHD-II criteria for ‘Typical aura without headache’ (code 1.2.3). Magnetic resonance imaging scan of the brain did not reveal any abnormalities. Extensive blood testing was normal. Factor V Leiden (R506Q) and prothrombin (G20210A) gene mutations were absent. This is the first case, to our knowledge, describing the conversion of MO into typical aura without headache after antihypertensive therapy. Mammalian brain harbours a renin-angiotensin system [3] and angiotensin receptors (AT1, AT2 and AT4) are plentiful in human brain. Our case report suggests a possible Conversion from migraine without aura to typical aura without headache after irbesartan