English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

The fractality of human gait, namely, the long-range correlation thatcharacterizes scale-free fluctuations of gait descriptors, such as the strideintervals during steady-state walking, depends on the well-tuned organizationof the sensorimotor controller. Gait fractality is apparent in healthy youngadults but tends to disappear in elderly individuals and neurological patients.Therefore, its partial loss may be indicative of pathological conditions.Despite its potential to be used as a dynamical biomarker for fall riskassessment, the mechanistic origin of gait fractality has been investigated byonly a few studies, and even less attention has been devoted to the linkbetween gait fractality and gait stability. Here, we propose a novelcomputational model of gait by addressing the flexibility-stability trade-offfirst, and then by showing that gait fractality is a natural consequence of thedeveloped control mechanisms, including phase resetting and intermittentcontrol, which supplement instability in a linear feedback controller operatedat stability's edge. The results suggest that pathological gait, characterizedby joint-rigidity and/or loss of fractality, may be caused by dysfunction insome of these mechanisms.

Phase resetting and intermittent control at critical edge of stability as major mechanisms of fractality in human gait cycle variability