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Atrial fibrillation (AF) represents the most common cardiac arrhythmia in daily clinical practice and substantially impacts affected patients by elevation of both morbidity and mortality. Previous investigations proved that inflammatory processes are closely linked to this multifactorial pathogenesis-especially autoreactive CD4 + CD28 null T cells received in-depth attention.

The impact of CD4+CD28null T lymphocytes on atrial fibrillation: a potential pathophysiological pathway