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Variation at the apolipoprotein E locus on chromosome 19 plays a role in more cases of Alzheimer’s disease than does any other identified genetic determinant. We have previously reported the isoform-specific interaction of native human apolipoprotein E (APOE, gene; apoE, protein) ε3 with the amyloid-ß peptide, Aß1−40, the major component of the cerebral amyloid deposits that appear to cause Alzheimer’s disease.

molecular medicine

Human Cerebrospinal Fluid Apolipoprotein E Isoforms are Apparently Inefficient at Complexing with Synthetic Alzheimer’s Amyloid-β Peptide (Aß1−40) in vitro