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Amyloid-β (Aβ) accumulates in plaques and as cerebral amyloid angiopathy (CAA) in the brains of both Alzheimer’s disease (AD) patients and transgenic AβPPswe/tg2576 (tg2576) mice. Increasingly, evidence in humans and mice shows this process to be modulated by apolipoprotein E (apoE).

Elevated Aβ and Apolipoprotein E in AβPP Transgenic Mice and Its Relationship to Amyloid Accumulation in Alzheimer’s Disease