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fall over a 70-80 rain interval to 4.7 mmol/1 or above (average fall rate 0.19_+0.05 mmo1-1.min -1, lower levels being prevented by prompt glucose infusion). No normal subjects released growth hormone during the study. In contrast, six of the nine diabetic patients (three Type I and three Type 2; Figs. 1 and 2) released growth hormone during or shortly after the fall in glucose concentration. These results suggest that rapidly declining hyperglycaemia may stimulate the release of growth hormone in Type 1 and non-obese Type 2 diabetic patients. This response is not seen in normal subjects. The release of growth hormone is likely to be induced by the rapid fall in glucose. Reaching a threshold level of plasma glucose probably acts as a trigger for growth hormone release, reflecting an upward resetting of the glucostat in diabetic subjects [6], although this cannot be substantiated by these data. In view of the debated role of growth hormone in the pathogenesis of diabetes mellitus and its chronic complications, the abnormal release of growth hormone during a decrease in plasma glucose level in diabetes may have clinical significance. Intermittent elevations of growth hormone may also contribute to the problems of diabetic control.

No linkage between HLA and maturity onset hyperglycaemia in the young