English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Plus annual

Presents the access of premium content as premium feature

Premium Content

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools annual

Global: Zendy Free Plan

Global: Zendy Tools monthly

Global: Zendy Plus monthly

infection 1 month earlier. Visual acuity was 20/80 OD and 20/30 OS. Ophthalmoscopy showed scattered active foci of whitening of the outer retina in both eyes (Figure 1A and B). Fluorescein angiography showed hypofluorescence during the early phases of the angiogram with late staining of the lesions (Figure 1C and D). A diagnosis of APMPPE was made, but no treatment was given. During the next 3 months, his symptoms gradually resolved and visual acuity returned to 20/20 OU. The patient had no additional eye symptoms until 16 years later, when he noted the onset of a temporal scotoma and peripheral shimmering in the left eye. Best corrected visual acuity was 20/25 OD and 20/30 OS. The vitreous body was clear. The macular area showed the old lesions of APMPPE (largely unchanged and without the development of significant atrophy). However, both eyes showed atrophic punched-out chorioretinal scars (Figure 2) in the macula, midperiphery, and far periphery. The appearance of these scars and the patient’s symptoms were consistent with a diagnosis of MFC.

Normalization of Upper Eyelid Height and Contour After Bony Decompressionin Thyroid-Related Ophthalmopathy: A Digital Image Analysis