Open AccessInhibition of KL-6/MUC1 glycosylation limits aggressive progression of pancreatic cancer
Author(s) -
Huanli Xu
Publication year - 2014
Publication title -
world journal of gastroenterology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.427
H-Index - 155
eISSN - 2219-2840
pISSN - 1007-9327
DOI - 10.3748/wjg.v20.i34.12171
Subject(s) - pancreatic cancer , vimentin , muc1 , cancer research , glycosylation , tunicamycin , metastasis , epithelial–mesenchymal transition , cadherin , tumor progression , immunohistochemistry , adenocarcinoma , pancreatic tumor , biology , cancer , pathology , medicine , cell , apoptosis , biochemistry , unfolded protein response
To evaluate the significance of KL-6/MUC1 (a type of MUC1) glycosylation in pancreatic cancer progression.