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Subclinical ultrasonographic abnormalities of the suspensory ligament branches in National Hunt racehorses
Author(s) -
Fairburn A. J.,
Busschers E.,
Barr A. R. S.
Publication year - 2017
Publication title -
equine veterinary journal
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.82
H-Index - 87
eISSN - 2042-3306
pISSN - 0425-1644
DOI - 10.1111/evj.12639
Subject(s) - suspensory ligament , subclinical infection , medicine , ligament , ultrasonography , anatomy , biology , veterinary medicine , radiology , pathology
Summary Background Suspensory ligament branch ( SLB ) desmopathy is a common cause of lameness and an important cause of lost training in the Thoroughbred racing industry. Studies have assessed the impact of insertional injuries of the SLB on the careers of flat racehorses and established the prevalence of subclinical ultrasonographic SLB abnormalities in this population, but little work has investigated SLB injury in National Hunt ( NH ) racehorses. Objectives To investigate the prevalence of subclinical ultrasonographic SLB abnormalities in NH racehorses with no clinical signs or history of SLB injury and to establish the cross‐sectional area ( CSA ) of SLB s in this population. Study design Cross‐sectional study using data collected from horses on an NH yard. Methods Ultrasonographic examination of forelimb SLB s in 62 horses on a single NH yard was performed. Images were graded according to a previously reported system. CSA measurements were obtained from transverse images. Results Nineteen of 62 horses had at least one SLB with grade 2 ultrasonographic abnormalities. Grade 2 ultrasonographic abnormalities occurred more frequently in the medial than the lateral SLB (P = 0.05). The medial SLB insertional CSA was significantly larger (P<0.001) than that of the lateral SLB . Main limitations Length of time on the yard (and therefore available veterinary history) is variable in this population. Conclusions One in three NH racehorses without history or clinical signs of SLB injury had at least one SLB with a grade 2 ultrasonographic abnormality. The medial branch was over‐represented. The medial SLB insertional CSA is larger than the lateral and thus comparison with the corresponding branch in the contralateral limb is recommended to avoid misdiagnosis of medial SLB enlargement.
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