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Alterations in NKG2A and NKG2C Subsets of Natural Killer Cells Following Epstein–Barr Virus Reactivation in CTLA4Ig-based Haploidentical Transplantation Is Associated With Increased Chronic Graft-Versus-Host Disease
Author(s) -
Sarita Rani Jaiswal,
Prakash Bhakuni,
Gitali Bhagwati,
Hema Malini Aiyar,
Aditi Chakrabarti,
Suparno Chakrabarti
Publication year - 2020
Publication title -
transplantation
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.45
H-Index - 204
eISSN - 1534-6080
pISSN - 0041-1337
DOI - 10.1097/tp.0000000000002941
Subject(s) - immunology , medicine , transplantation , graft versus host disease , lymphoproliferative disorders , cyclophosphamide , incidence (geometry) , virus , lymphoma , chemotherapy , physics , optics
The impact of newer approaches to haploidentical transplantation on Epstein-Barr virus (EBV) is largely unknown.

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