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P4‐308: CSF α‐synuclein, BETA‐AMYLOID 1–42 and tau proteins: Relationship to clinical parameters in early Parkinson's disease
Author(s) -
Kang Ju Hee,
ChenPlotkin Alice,
Irwin David,
Siderowf Andrew,
Caspell Chelsea,
Coffey Chris,
Taylor Peggy,
Frasier Mark,
Marek Kenneth,
Mollenhauer Brit,
Trojanowski John,
Shaw Leslie
Publication year - 2012
Publication title -
alzheimer's and dementia
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 6.713
H-Index - 118
eISSN - 1552-5279
pISSN - 1552-5260
DOI - 10.1016/j.jalz.2013.08.089
Subject(s) - medicine , biomarker , parkinson's disease , gastroenterology , oncology , pathophysiology , logistic regression , pathology , psychology , disease , chemistry , biochemistry
dorsolateral prefrontal cortex (figure 2). 2) shown differentiation in contrast of structural elements in this region indicating our ability to see vasculature and the myelinated axon bundles associated with neuronal columns (figures 2, 3, 4). Conclusions: Fine SA has been shown to enable in vivo probing of neuronal fibre bundle structure within the human cortex. Our initial results indicate this assessment technique is capable of monitoring age and pathology related changes in neuronal organisation within the cortex.