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Lab A complete blood count, basic metabolic profile, hepatic and lipid panels were within normal limits and we excluded systemic involvement. A bone density study showed osteoporosis. Serum tryptase levels, 24 hour urinary N-methylhistamine, N-methylimidazoleacetic acid and prostaglandin D2 metabolites excretion werewithin normal limits. Skin biopsy cofirmed the diagnosis of generalized cutaneous mastocytosis (Urticaria pigmentosa) (Fig. 2A-D). The patient left the Dermatology Unit with no medication, but she called us, a few weeks later for a new appointment. She described and we confirmed the dissapearance of the cutaneous lesions during the last weeks, while she was hospitalised for a hip fracture and treated with Enoxaparinum 40mg s.c/daily for 14 days. The patient refused a new biopsy and we saw her again three months later, she again showed the characteristic brownishred skin lesions of Urticaria pigmentosa, exactly as at the first appointment. The lesions had begun to appear very soon after she had stopped taking Enoxaparinum (Fig. 3). ADULT URTICARIA PIGMENTOSA WITH TRANSITORY DISAPPEARANCE OF LESIONS DURING ENOXAPARINUM TREATMENT

Adult urticaria pigmentosa with transitory disappearance of lesions during enoxaparinum treatment