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50 complete standard Tekland test diet ad libitum (Tekland, Madison, WI, USA) with 0.1% DDC or 2.5% GF for 1 year and then sacrificed. Control mice were fed the basal diet without DDC. As soon as the mice were killed, their liver was removed. Portions of the liver were fixed in formalin and embedded in paraffin for light microscopic analysis by hematoxylin-eosin and gamma glutamyl transpeptidase (GGT) was accessed by histochemistry. Figure 1 illustrates an HN from a mouse fed DDC for 1 year. The tumor is devoid of protoporphyrin and is surrounded by normal liver parenchyma filled with protoporphyrin deposits. Figure 2 shows hyperplastic foci stained red for GGT, a marker for HN, showing that the precursor lesion is devoid of protoporphyrin deposits. HN or foci are generated during the process of liver cancer development in response to chemical carcinogens or dietary Preneoplastic foci in mice fed diethyl 1, 4-dihydro, 1, 4, 6-trimethyl 3, 5-pyridine decarboxylate are resistant to protoporphyrin accumulation