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Tinnitus is defined as an auditory phantom perception without a corresponding external sound source. Tinnitus is one of the most common otologic symptoms, affecting 10–15% of people (1-3). One potential mechanism of tinnitus is the alteration of perception in neural auditory pathways. Therefore, tinnitus is often the focus of neuroscience studies involving functional MRI (fMRI). In the last decade, fMRI in tinnitus has focused on task-based neuronal stimulations using auditory and somatic modulation. One limitation of task-based fMRI in tinnitus is the bias from modulation processing. Because auditory or somatic modulation in a tinnitus patient is often too impaired to correctly perform the requested task, task-based fMRI studies in tinnitus patients are difficult to generalize. Contrary to task-based fMRI, resting-state fMRI (rs-fMRI) is a relatively novel technique that has the potential to overcome many of these limitations. Because tinnitus involves consistently abnormal intrinsic neural activation, it is a suitable subject for rs-fMRI. Several previous studies demonstrated alterations of resting-state functional connectivity (FC) in tinnitus using magnetoencephalography (4, 5). These studies reported different activities and connectivities in the auditory cortex networks of the tinnitus groups compared to the control groups. Additional Change in Functional Connectivity in Tinnitus and its Relation with Tinnitus Laterality