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Our understanding of the pathophysiology of acquired thrombotic thrombocytopenic purpura has improved remarkably over the past ten years. Nonetheless, recent studies raise new questions concerning ADAMTS13 activity, anti-ADAMTS13 autoantibodies and risk of recurrent disease. These questions likely far outweigh the substantial knowledge gained. See related article on page 232.

Acquired thrombotic thrombocytopenic purpura: ADAMTS13 activity, anti-ADAMTS13 autoantibodies and risk of recurrent disease