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Advances in the understanding of the pathogenesis of MPGN have resulted in a new classification of MPGN based on pathogenetic pathways. In this system, MPGN is nowadays classified as being mediated by immune complexes, by complement dysregulation or rarely by mechanisms not involving immunoglobulin or complement deposition, such as endothelial injury. Autoimmune diseases, monoclonal gammopathy/dysproteinemias, and chronic infections are common causes of immune -complex -mediated MPGN.4 The clinical presentation is diverse, with variable degrees of kidney impairment; hypertension may or may not be present. The diagnosis is made by renal biopsy.2

Cryoglobulinemic HCV-related membranoproliferative glomerulonephritis with TMA – secondary HUS or infection-triggered aHUS?