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hypothalamus. The vasopressin gene resides on chromosome 20 and consists ofexons A, B and C. Exons A, B and C include coding regions of three types of mRNAtranslating signal peptide and vasopressin, neurophysin II and glycoprotein, respectively. The pre-prohormone of vasopressin, the precursor of vasopressin during its biosynthesis pathway, consists of these three components ( 1). The inborn error of hypothalamic vasopressin synthesis was initially elucidated in rats with hereditary hypothalamic diabetes insipidus (2) , the Brattleboro strain. In these animals the vasopressin gene has a single base deleted from vasopressin-associated neurophysin-coding region of exon B. This causes a frame shift and eliminates a stop codon, resulting in a vasopressin precursor with an altered Cterminal region. Despite the intact signal and vasopressin

Molecular Aspects of the Pathogenesis Involved in the Development of Familial Central Diabetes Insipidus