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Tubular dysfunctions of renal homograft cases have been reported from several aspects (I). However, chronic hypophosphatemia of the kidney transplant recipient is a relatively new topic (4). It is encountered frequently and is considered to be very important in differential diagnosis of persistent hyper­ parathyroidism. We studied this chronic hypophosphatemia in our intrafamiliar renal homograft recipients.

Chronic hypophosphatemia in the renal homograft recipient.