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Modern diabetology has undergone a continual change ranging from the biological definition of different clinical diagnoses to anatomo-pathological classifications, going through panoply of radiological examinations. It is in this sense that introduced a new histopathological classification based on the different components of nephrons (not only glomerular lesions but also interstitial, vascular and tubular lesions).1,2 All this has been accompanied by a change in the concept and denomination of the pathology from diabetic nephropathy to diabetic kidney disease.3 If the mechanism explaining the aggravation of Diabetic kidney disease in cases of glycemic imbalance or association with high blood pressure (hyperactivity of the cotransporter SGLT2 as well as hyperfiltration which exhausts the nephron in the long course).4 This is not yet the case for the heterogeneous group of non-diabetic nephropathies for lack of histological evidence based on invasive diagnostic tools such as biopsy which can be supplanted by the new biological and radiological tools. To my knowledge no study has been done until to now on the impact of non-diabetic nephropathy on prognosis of coexisting diabetic nephropathy) because contrary to what some people think the effect is not necessarily an aggravation, which can be simulated to the influence of refractory vices on Diabetic retinopathy (myopia protects against Diabetic retinopathy (DR) and hyperopia aggravates it).5

Diabetic kidney disease: diabetic and non-diabetic nephropathy, Updates 2019