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fdx1b null-allele zebrafish larvae are impaired in cortisol synthesis, cortisol regulated gene expression and in their stress response 0.1 mm Significant gaps remain on the understanding of the in vivo impact of cortisol deficiency on metabolic pathways during embryonic development. Herein, we present a newly established cortisol deficient zebrafish mutant line in order to investigate into the pathogenic effects of cortisol deficiency in vivo.

Exploring metabolomic changes due to cortisol deficiency in early development using the ferredoxin (fdx1b) null-allele zebrafish