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muscle sympathetic nerve activity (MSNA), in spite of insulin resistance (Seravalle et al., Clin Endocrinol 77:262, 2012). Our data pointed to a phenomenon mediated by hypoleptinaemia rather than a direct action of the GH-IGF-I system. Aim: It has been shown that centrally administered somatostatin (SS) inhibits peripheral sympathetic outflow in rodents (Rettig et al., Am J Physiol 257:R588, 1989). Based on the above, we elected to study MSNA in acromegalic patients before and during treatment with SS analogues (SSA)

Effects of somatostatin analogues on muscle sympathetic nerve activity in acromegaly