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l Introduction Beta-mimetic drugs are currently used in obstetrics to treat several disorders: Threatened premature labor, hyperdynamic labor, or acute fetal distress [2, 5, 6, 7, 8, 12]. Nevertheless the effectiveness o£ this treatment may be limited by the fact that the Stimulation of the uterine receptors i s not specific and is accompanied by the Stimulation of the other beta-receptors distributed throughout the whole anatomy. For this reason, the desirable Inhibition of the uterine activity is accompanied by the appearance of the undesirable side-effects. The most evident among them are those related to the Stimulation of the cardiovascular beta-receptors (tachycardia, etc.). On the other hand, the Stimulation of the metabolic beta-receptors, leads, as can be exepted, to an jncrease in the levels of blood glucose [8], 11. By avoiding the cardiovascular and/ the metabolic effects of these drugs they could be used in those cases in which a preexisting pathology (heart disease, diabetes, etc.) might result in a serious deleterious Situation. In some normal patients the need of high doses to obtain a suitable uterine effect, could result in a greater than usual development of the side-effects, thus making it desirable in these normal patients to supress them, at least> to some extent. An ideal Situation would be the possibility of obtaining specific stimulators for the receptor sites of different organs. But, up to the present time, nothing indicates a qualitative differentation Curriculum vitae

An attempt at blocking the cardiovascular and metabolic side-effects of beta-mimetic drugs during labor