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ultraviolet ti|ht (UV) is the mosl important cause of human skin cancers. UV resuits in carcinogenesis through direct effects on the target cell aid indirect effects on the host (immunosuppression). Recen! studies have begun to uncover the molecular mechanisms involved. Here, we discuss recent findings and developments in thc molecular biology of UV-carcinogenesis in the skin, by focusing on DNA damage and DNA repair, the p53 tumor suppressor gene, apoptosis, the patched gene and telomerase activity.

Molecular pathological changes induced by ultraviolet light during the development of skin cancer