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(0.2±0.8% vs. 1.3±2.8%, P=0.006) and the frequency of stenttreated lesion with any malapposed struts (n=6, 11% vs. n=14, 27%, P=0.028) were meaningfully lower in EES compared with BES. Therefore, the vascular healing process is more favorable with EES than with BES. However, unmet questions remain. First, there was no initial imaging by OCT just after DES implantation, so the authors could not exclude a difference in stent malapposition at the index PCI. Second, the vascular healing process is progressive in the case of BES.7 Therefore, the possibility of a difference in the period of vascular healing according to the type of DES remains. Third, although OCT offers higher resolution imaging than any other imaging modality, it does not have a sufficient resolution to detect endothelialization forming a 5-μm thick cellular monolayer. Fourth, despite much OCT data being published, there are few reports that link OCT data with clinical outcomes. Not withstanding these limitations, the present report supports the results that EES are superior to BES in terms of 1-year definite stent thrombosis.6,8 Unmet problems remain: How does the vascular healing process with BES progress over time? How do we decide the duration of dual antiplatelet therapy according to the type of DES? In the future, both of these critical points should be investigated.

What Is the Optimal Time to Estimate the Final Vascular Response to a Drug-Eluting Stent by Optimal Coherence Tomography?