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Microvascular sequestration of  Plasmodium falciparum  underlies cerebral malaria. Despite suggestive  ex vivo  evidence, this phenomenon has not been convincingly demonstrated in coma complicating  Plasmodium vivax  malaria. Severely-ill Papua New Guinean children with mixed  P. falciparum/P. vivax  infections are more likely to develop cerebral malaria and die than those with  P. falciparum  alone, possibly reflecting  P. vivax  sequestration. Nested PCR was performed on  post mortem  brain tissue from three such children dying from cerebral malaria due to mixed-species infections. No  P. vivax  DNA was detected. These findings do not support the hypothesis that  P. vivax  sequestration occurs in human brain.

A histopathologic study of fatal paediatric cerebral malaria caused by mixed Plasmodium falciparum/Plasmodium vivax infections