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RESULTS: In the observational analyses, birthweight showed inverse associations with systolic and diastolic blood pressure (β, −0.83 and −0.26; per raw unit in outcomes and SD change in birthweight; 95% confidence interval [CI], −0.90 to −0.75 and −0.31 to −0.22, respectively), T2D (odds ratio, 0.83; 95% CI, 0.79–0.87), lipid-lowering treatment (odds ratio, 0.84; 95% CI, 0.81–0.86), and coronary artery disease (hazard ratio, 0.85; 95% CI, 0.78–0.94), whereas the associations with adult body mass index and body fat (β, 0.04 and 0.02; per SD change in outcomes and birthweight; 95% CI, 0.03–0.04 and 0.01–0.02, respectively) were positive. The Mendelian randomization analyses indicated inverse causal associations of birthweight with low-density lipoprotein cholesterol, 2-hour glucose, coronary artery disease, and T2D and positive causal association with body mass index but no associations with blood pressure.

Correction to: Birthweight, Type 2 Diabetes Mellitus, and Cardiovascular Disease: Addressing the Barker Hypothesis With Mendelian Randomization