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The cardiac inwardly rectifying K(+) current (I(K1)) plays a critical role in modulating excitability by setting the resting membrane potential and shaping phase 3 of the cardiac action potential.

Nitric Oxide Increases Cardiac I K1 by Nitrosylation of Cysteine 76 of Kir2.1 Channels

Nitric Oxide Increases Cardiac I _K1 by Nitrosylation of Cysteine 76 of Kir2.1 Channels