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KCNQ1 and KCNE1 assemble to form the slow delayed rectifier ( I  Ks ) channel critical for shortening ventricular action potentials during high β-adrenergic tone. However, too much I  Ks under basal conditions poses an arrhythmogenic risk. Our objective is to understand how adult ventricular myocytes regulate the I  Ks amplitudes under basal conditions and in response to stress.

circulation. arrhythmia and electrophysiology

Adult Ventricular Myocytes Segregate KCNQ1 and KCNE1 to Keep the I Ks Amplitude in Check Until When Larger I Ks Is Needed