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This study used monophasic action potential (MAP) to examine the effect of nicorandil, a K+ channel opener, on repolarization abnormalities induced by epinephrine in the LQT1 form of congenital long-QT syndrome in which the KvLQT1 mutation underlies the defect in the channel responsible for the slowly activating component of the delayed rectifier potassium current.

Improvement of Repolarization Abnormalities by a K + Channel Opener in the LQT1 Form of Congenital Long-QT Syndrome

Improvement of Repolarization Abnormalities by a K ⁺ Channel Opener in the LQT1 Form of Congenital Long-QT Syndrome