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Intracellular calcium overload is believed to play an important role in development of reperfusion arrhythmias. Dipyridamole, an inhibitor of cellular uptake of adenosine, may prevent or terminate reperfusion arrhythmias by reducing intracellular calcium overload.

Antiarrhythmic Efficacy of Dipyridamole in Treatment of Reperfusion Arrhythmias