English (United Kingdom)

https://curated-unify.zendy.io/wp-json/zendy-region/v1/featured_content/oa?rat=en

https://curated-unify.zendy.io/wp-json/zendy-region/v1/highlighted_journal/

Global: Zendy Tools annual

Presents the keyphrase highlighting as premium feature

Keyphrase Highlighting

Presents the summarisation as premium feature

Summarisation

Insights

Presents the pdf analysis as premium feature

PDF Analysis

Presents the zaia usage as premium feature

ZAIA

Global: Zendy Tools monthly

Global: Zendy Plus monthly

Presents the access of premium content as premium feature

Premium Content

Global: Zendy Plus annual

Global: Zendy Free Plan

To investigate the efficacy of phage supplementation in reducing pathogen numbers, mice were treated via oral gavage with a  Listeria monocytogenes  phage preparation (designated ListShield) before being orally infected with  L. monocytogenes . The concentrations of  L. monocytogenes  in the liver, spleen, and intestines were significantly lower ( P  < .05) in the phage-treated than in the control mice. Phage and antibiotic treatments were similarly effective in reducing the levels of  L. monocytogenes  in the internal organs of the infected mice. However, the significant weight loss detected in the control and antibiotic-treated groups was not observed in the infected, ListShield-treated mice. Long-term (90 days), biweekly treatment of uninfected mice with ListShield did not elicit detectable changes in the microbiota of their large intestines or deleterious changes in their health. Our data support the potential feasibility of using bacteriophages to control proliferation of  L. monocytogenes  in mice without affecting commensal microbiota composition.

international journal of microbiology

Bacteriophage Administration Reduces the Concentration of<i>Listeria monocytogenes</i>in the Gastrointestinal Tract and Its Translocation to Spleen and Liver in Experimentally Infected Mice

Bacteriophage Administration Reduces the Concentration ofListeria monocytogenesin the Gastrointestinal Tract and Its Translocation to Spleen and Liver in Experimentally Infected Mice