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We show that a simple biophysically based electron-conformational model ofRyR channel is able to explain and describe on equal footing the oscillatoryregime of the heart's cell release unit both in sinoatrial node (pacemaker)cells under normal physiological conditions and in ventricular myocytes underCa$^{2+}$ SR overload.

Electron-conformational transformations in nanoscopic RyR channels govern both the heart's contraction and beating