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DuringSalmonella enterica serovar Typhimurium infection, host inflammation alters the metabolic environment of the gut lumen to favor the outgrowth of the pathogen at the expense of the microbiota. Inflammation-driven changes in host cell metabolism lead to the release ofl -lactate and molecular oxygen from the tissue into the gut lumen.

Host-Derived Metabolites Modulate Transcription of Salmonella Genes Involved in l -Lactate Utilization during Gut Colonization