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We read with interest the recent article, “Alexander disease mutant glial fibrillary acidic protein compromises glutamate transport in astrocytes,” by Tian et al (1). The authors documented impairment of glutamate transport in aberrant astrocytes bearing mutant glial fibrillary acidic protein (GFAP) that may play an important role in the pathogenesis of oligodendrocyte and neuronal degeneration in Alexander disease (AxD). …

journal of neuropathology and experimental neurology

Correspondence Regarding: Alexander Disease Mutant Glial Fibrillary Acidic Protein Compromises Glutamate Transport in Astrocytes J Neuropathol Exp Neurol 2010;69:335–45