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OBJECTIVETumor stem cells (CSCs) are considered to be involved in tumor relapse. Mammalian Target of Rapamycin Complex 1 (mTORC1) plays an important role in tumor development and growing. However, the significance of mTORC1 in CSCs remains controversial. This study aims to provide more information for further understanding the mechanisms in down regulating mTORC1 activity in glioma stem cells (GSC).

TBIO-02. INHIBITION OF mTORC1 THROUGH AMINO ACID TRANSPORTER CD98/LAT1 MAINTAINS THE STEMNESS OF GLIOMA STEM CELL