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Both a decrease in bone formation and the skeletal consequences of secondary hyperparathyroidism have been implied in the pathogenesis of age-related femoral neck osteoporosis. However, studies using biochemical indices of bone remodeling in hip fracture patients have yielded conflicting results. Similarly, secondary hyperparathyroidism has not been a consistent finding in this population. Some of these inconsistencies might reflect differences in the assays used as well as in the timing of the sampling. Moreover, measurements were mostly performed in a limited number of patients. In this regard, the aim of the present study was to analyze potential alterations in bone metabolism in a large population of elderly hip fracture patients.

the journals of gerontology. series a, biological sciences and medical sciences

Calciotropic Hormones and Markers of Bone Remodeling in Age-Related (Type II) Femoral Neck Osteoporosis: Alterations Consistent with Secondary Hyperparathyroidism-Induced Bone Resorption