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The length of the cell cycle delay imposed by the spindle checkpoint, also referred to as the spindle checkpoint strength, is controlled by the number of unattached kinetochores, cell volume, and cell fate. We show that PCH-2, a highly conserved AAA-ATPase, controls checkpoint strength during early embryogenesis in C. elegans.

The conserved AAA-ATPase PCH-2TRIP13regulates spindle checkpoint strength

The conserved AAA-ATPase PCH-2^TRIP13regulates spindle checkpoint strength