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In this issue of The Journal of Experimental Medicine , Tuohy et al. study the spreading of the autoimmune response to myelin antigens in experimental autoimmune encephalomyelitis (EAE) and its likely human counterpart, multiple sclerosis (MS) (([1][1])). They demonstrate that as disease develops in

Absence of “Original Antigenic Sin” in Autoimmunity Provides an Unforeseen Platform for Immune Therapy