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Our modification of the assay for the inhibitor of factor Stuart activated (Anti-Xa) was evaluated together with the antiactivator of plasminogen (Antiplasm.) and the nature of the fibrinogen degradation products (FDP) as a test of the risk for thrombotic disorders. Thirty patients with acute myocardial infarction (AMI), twenty eight in the chronic phase (CMI) and thirty healthy controls (C) were studied. The age range for the three groups was 45-85 with a mean age of 57-62. The activity of Anti-Xa in C was 46.7 U/ml (± SD 11.5); 31.5 U/ml (± SD 16.5) with p&lt;0.001 for the AMI group and 42.1 u/ml (± SD 17.9) for the GMI group. Very high activity for Antiplasm. in the AMI group was found (11 min ± SD 7.1) with p &lt; 0.001 for a C value of 3 min 36 sec (± SD 1.2). For the CMI group the value was 6 min 11 sec (± SD 3.3) and p &lt; 0.001. The FDP were elevated in the AMI group with a mean value of 15.5 polimerization inhibition units (PIU/ml) being O PIU/ml the C value. The CMI group had a mean value of 4.3 PIU/ml. The ratio between PIU/ml and µg/ml of the FDP indicated products of “early” nature in AMI and “late” type in CMI, The findings mentioned above were similar when the patients received anticoagulants except for the nature of FDP that was changed to the “late” type in both groups. The simultaneous determination of Anti-Xa, Antiplasm. and the nature of the FDP can be applied to other pathologies. The comparison of these parameters may indicate the oncoming of an acute thrombotic episode and can be useful in evaluating the status of the cardiovascular system during cardiac rehabilitation.

Natural Plasma Inhibitors in Individuals with High Risk for Thrombotic Disease