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The aim of this study was to explore the cerebral distribution of the tau-specific PET tracer [(18)F]THK5317 (also known as (S)-[(18)F]THK5117) retention in different stages of Alzheimer's disease; and study any associations with markers of hypometabolism and amyloid-beta deposition.

Imaging in-vivo tau pathology in Alzheimer’s disease with THK5317 PET in a multimodal paradigm