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Endogenous γ-aminobutyric acid A receptor (GABA A R)-active neurosteroids (e.g., allopregnanolone) regulate central nervous system excitability and many physiological functions, so fluctuations are implicated in several neuropsychiatric disorders. Pertinently, evidence supports an inverse relationship between endogenous GABA A R-active neurosteroid levels and behavioral changes in excitability during ethanol withdrawal (WD).

Ethanol withdrawal-induced dysregulation of neurosteroid levels in plasma, cortex, and hippocampus in genetic animal models of high and low withdrawal